Original articleNeutrophil expression of CD11b/CD18 and IL-8 secretion during normothermic cardiopulmonary bypass
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2011, BiomaterialsCitation Excerpt :In the present studies we showed, in Chandler Loop Apparatus studies, that CD47 functionalized surfaces were superior to PMEA and control surfaces with respect to reducing cellular attachment and expression of CD18. The reduction in CD18 surface expression is important as CD18 expression has been associated with adverse outcomes following clinical procedures, such as CPB, where there is extensive interactions between blood and synthetic surfaces [24,35]. It has been previously observed that CD47-SIRP interactions occur in the presence of opsonins [7,36,37], thus any effects of adsorbed proteins on CD47 modified surfaces should be minimal with respect to its anti-inflammatory capacity.
The unwanted response to cardiac surgery: Time for a reappraisal?
2003, Journal of Thoracic and Cardiovascular SurgeryCitation Excerpt :The current study by Schmartz suggests the peak in interleukin 8 (IL-8) concentration is some 4 hours after bypass. This timing is supported by the data of Paugam and colleagues12 after normothermic bypass. However, the majority of studies have measured the peak in IL-8 concentration at the end of surgery or shortly after the release of the aortic crossclamp.
Hypothermia augments polymorphonuclear leukocyte degranulation and interleukin-8 production from human umbilical vein endothelial cells and increases lipopolysaccharide-induced polymorphonuclear leukocyte-endothelial cell interaction when followed by normothermia
2002, Journal of Cardiothoracic and Vascular AnesthesiaCitation Excerpt :In particular, IL-6 and IL-8 are most frequently detectable in patients undergoing CPB.3-9 Because PMN activation plays an important role in the development of organ injury associated with an acute inflammatory response, it was hypothesized that IL-8 is an important proinflammatory cytokine in patients in whom CPB was performed and who developed a subsequent organ injury.4,6-8 This hypothesis led the authors to investigate the effect of hypothermia on IL-8-induced PMN activation and on LPS-induced IL-8 production from HUVEC.
The role of MyD88- and TRIF-dependent signaling in monophosphoryl lipid A-induced expansion and recruitment of innate immunocytes
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