Neuron
ArticleNerve growth factor induces a gene homologous to the glucocorticoid receptor gene
References (31)
- et al.
Establishing homologies in protein sequences
Meth. Enzymol.
(1983) - et al.
Functional domains of the human glucocorticoid receptor
Cell
(1986) Point mutations define a sequence flanking the AUG initiator codon that modulates translation by eukaryotic ribosomes
Cell
(1986)- et al.
Genetic complementation of a glucocorticoid receptor deficiency by expression of cloned receptor cDNA
Cell
(1986) - et al.
A conserved AU sequence from 3' untranslated region of GM-CSF mRNA mediates selective mRNA degradation
Cell
(1986) - et al.
Cloning of human mineralocorticoid receptor complementary DNA: structural and functional kinship with the glucocorticoid receptor
Science
(1987) - et al.
Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease
Biochemistry
(1979) - et al.
Is actin a transcription initiation factor for RNA polymerase B?
EMBO J.
(1984) - et al.
A technique for radiolabeling DNA restriction endonuclease fragments to high specific activity
Anal. Biochem.
(1984) - et al.
Human oestrogen receptor cDNA: sequence, expression and homology to v-erb-A
Nature
(1986)
Effect of protein synthesis inhibitors on growth factor activation of c-fos, c-myc, and actin gene transcription
Mol. Cell. Biol.
Target cells, biological effects, and mechanism of action of nerve growth factor and its antibodies
Annu. Rev. Pharmacol. Toxicol.
A new method for purifying lambda DNA from phage lysates
DNA
Unidirectional digestion with exonuclease III creates targeted breakpoints for DNA sequencing
Gene
Constructing and screening cDNA libraries in λgt10 and λgtl1
Cited by (572)
Fragment-based discovery of orphan nuclear receptor Nur77/NGFI-B ligands
2022, Bioorganic ChemistryMinireview: What is Known about SUMOylation Among NR4A Family Members?
2021, Journal of Molecular BiologyCitation Excerpt :NUR77 (NR4A1, NGFI-B, TR3), NURR1 (NR4A2) and NOR-1 (NR4A3), members of the NR4A subfamily of nuclear receptors,1–3 are transcription factors that participate in multiple essential cellular functions such as cell cycle and apoptosis, lipid metabolism, inflammation, carcinogenesis, vascular and neuronal functions, among others.4–8
Negative feedback by NUR77/Nr4a1 restrains B cell clonal dominance during early T-dependent immune responses
2021, Cell ReportsCitation Excerpt :Here, we test whether the orphan nuclear receptor NUR77/Nr4a1 may restrain immunodominance in the early GC by mediating a negative feedback loop downstream of the B cell receptor (BCR). NUR77 is rapidly upregulated by antigen receptor signaling and is thought to function as a ligand-independent transcription factor (Hazel et al., 1988; Milbrandt, 1988; Mittelstadt and DeFranco, 1993; Winoto and Littman, 2002). In T cells, NUR77 and its family members mediate thymic negative selection (Calnan et al., 1995; Liu et al., 1994; Woronicz et al., 1994), anergy (Liu et al., 2019), and exhaustion (Chen et al., 2019) and reinforce regulatory T cell (Treg) identity (Sekiya et al., 2013).
Anaesthesia-induced Changes in Genomic Expression Leading to Neurodegeneration
2024, CNS and Neurological Disorders - Drug Targets