<?xml version='1.0' encoding='UTF-8'?><xml><records><record><source-app name="HighWire" version="7.x">Drupal-HighWire</source-app><ref-type name="Journal Article">17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">SOLİM, LEVENT AKMAN</style></author><author><style face="normal" font="default" size="100%">GENCAN, İZGI AYÇIL</style></author><author><style face="normal" font="default" size="100%">ÇELİK, BEHIÇ</style></author><author><style face="normal" font="default" size="100%">ATAACAR, ALARA</style></author><author><style face="normal" font="default" size="100%">KOÇ, UMUT</style></author><author><style face="normal" font="default" size="100%">BÜYÜKÖREN, BEYZA</style></author><author><style face="normal" font="default" size="100%">GÜNGÖR, GIZEM</style></author><author><style face="normal" font="default" size="100%">İSBİR, SELIM</style></author></authors><secondary-authors></secondary-authors></contributors><titles><title><style face="normal" font="default" size="100%">Endothelial Lipase Gene Polymorphism (584 C/T) in Coronary Artery Patients Among a Turkish Population</style></title><secondary-title><style face="normal" font="default" size="100%">In Vivo</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2018</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2018-09-01 00:00:00</style></date></pub-dates></dates><pages><style  face="normal" font="default" size="100%">1105-1109</style></pages><doi><style  face="normal" font="default" size="100%">10.21873/invivo.11352</style></doi><volume><style face="normal" font="default" size="100%">32</style></volume><issue><style face="normal" font="default" size="100%">5</style></issue><abstract><style  face="normal" font="default" size="100%">Background/Aim: The endothelial lipase gene (LIPG) has a major role in regulating high density lipoprotein cholesterol (HDL-C), therefore this study investigated whether LIPG is associated with coronary artery disease (CAD) in a Turkish population. Materials and Methods: The LIPG (584 C/T) mutation was analyzed in 74 CAD patients and 73 controls. Results: There was a significant difference between the two groups regarding the mutant T allele frequencies (χ2=0.456, p=0.020; 26.7% and 41.8% in patient and control groups, respectively) for 584 C/T. Even though the TT genotype was not significantly different, it had p=0.054 which supported our results. Conclusion: The endothelial lipase gene (584 C/T) T allele might be protective in association with coronary artery disease. Therefore, LIPG gene is related to risk for CAD in the Turkish population probably through altering HDL-C metabolism.</style></abstract></record></records></xml>